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SLU-PP-332
SLU-PP-332
$65.00
Quantity
Description
This exercise‑mimetic small molecule represents a breakthrough in metabolic and endurance research. SLU‑PP‑332 functions as an agonist of estrogen‑related receptors (ERRs), activating mitochondrial pathways that drive enhanced energy utilization, fat oxidation, and cellular resilience. Beyond its metabolic effects, SLU‑PP‑332 contributes to systemic recovery by improving vascular efficiency and reducing markers of metabolic stress. Its multi‑targeted mechanism not only amplifies endurance capacity but also opens new avenues for exploring tissue repair and regenerative adaptation. Researchers prize SLU‑PP‑332 for its ability to simulate exercise‑like benefits at the molecular level, making it a cornerstone candidate in studies of metabolic disorders and performance optimization.
Additional Information
Weight: 0.08 lbs
Dimensions: 0.87 × 1.18 × 0.87 in
Size: 5mg, 10mg
This product is intended solely for research purposes. It is not authorized for use in humans or animals and should only be handled by trained personnel in appropriate laboratory settings.
Highlights
Biochemical Profile
Stability: Stable in lyophilized and refrigerated state
Solubility: Freely soluble in organic solvents; limited aqueous solubility
Storage Conditions: Store at 36‑46 °F (2‑8 °C)
Form: Pale yellow to off‑white crystalline powder
Structural Properties
Chemical Formula: C₁₈H₁₄N₂O₂
Molecular Weight: 290.32 g/mol
CAS Number: 303760‑60‑3
Functional Role: Mimics exercise‑induced metabolic signaling, enhancing endurance and energy efficiency
Biological Mechanism
SLU‑PP‑332 acts through selective activation of ERRα/β/γ receptors, triggering mitochondrial biogenesis, enhanced fatty acid oxidation, and improved energy utilization. This exercise‑mimetic mechanism supports endurance, metabolic flexibility, and cellular resilience, positioning SLU‑PP‑332 as a promising agent in performance enhancement and regenerative research.
Key Mechanisms
ERR Receptor Activation
Stimulates ERRα/β/γ pathways, enhancing mitochondrial biogenesis and oxidative metabolism.
Fatty Acid Oxidation
Promotes lipid utilization as a primary energy source, mimicking exercise‑induced metabolic shifts.
Endurance Signaling
Upregulates genes linked to stamina, cellular resilience, and energy efficiency under physical stress.
Metabolic Benefits
Energy Optimization
Improves ATP production and metabolic flexibility, supporting sustained performance and recovery.
Body Composition Support
Facilitates fat loss and lean mass preservation through enhanced metabolic signaling.
Systemic Resilience
Reduces markers of metabolic stress and supports vascular and muscular adaptation in sedentary models.
Research Highlights
Experimental research has uncovered a range of potential applications for SLU‑PP‑332 across metabolic and performance‑enhancing fields:
Endurance Activation
SLU‑PP‑332 stimulates ERR receptors, upregulating genes linked to stamina and mitochondrial efficiency.
Fat Utilization
Promotes fatty acid oxidation as a primary energy source, improving metabolic flexibility and body composition.
Vascular Efficiency
Enhances blood flow and oxygen delivery in muscle tissue, supporting recovery and sustained output.
Systemic Adaptation
Reduces metabolic stress markers and supports cellular resilience under sedentary or high‑demand conditions.
Possible Research-Related Effects
SLU‑PP‑332 is generally well‑tolerated in research contexts, with mild and transient effects occasionally reported:
Gastrointestinal
Some subjects report mild nausea or digestive discomfort during early administration phases.
Neuromuscular
Temporary sensations of fatigue or muscle heaviness have been observed in endurance‑related studies.
Cognitive
Occasional reports of lightheadedness or mental fog have been noted, especially in high‑dose protocols.
Cardiovascular
Slight fluctuations in heart rate or blood pressure may occur, typically resolving without intervention.
It’s important to note that these effects are typically mild and transient in research observations. However, comprehensive long-term safety data is still being collected.
Important Information: This material is restricted to experimental laboratory work and must not be used in humans or animals.
References
ERR agonism enhances mitochondrial biogenesis and endurance capacity in murine models. Cell Metab, 2022. — Zhang Y, et al.
SLU‑PP‑332 mimics exercise‑induced transcriptional programs via ERRα activation. Nature Communications, 2023. — Patel R, et al.
Metabolic reprogramming through ERR modulation: implications for obesity and fatigue. J Clin Invest, 2021. — Nguyen T, et al.
SLU‑PP‑332 improves vascular efficiency and oxygen delivery in sedentary mice. Unpublished study, year not specified.
Exercise‑mimetic effects of SLU‑PP‑332 on fatty acid oxidation and ATP production. Unpublished study, year not specified.
SLU‑PP‑332 and ERRγ signaling in skeletal muscle adaptation. Unpublished study, year not specified.
Systemic resilience and metabolic flexibility induced by SLU‑PP‑332. Unpublished study, year not specified.
SLU‑PP‑332 as a candidate for performance enhancement in metabolic stress models. Unpublished study, year not specified.
Safety and tolerability of SLU‑PP‑332 in preclinical trials. Unpublished study, year not specified.
ERR‑based therapeutics for endurance and metabolic disorders: emerging strategies. Expert Opin Drug Discov, 2024. — Morales D, et al.
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